Home > Biological Science > Biotechnology > Volume-2 > Issue-4 > Anti-cancer Activity of Vitamin-C Against Leukemia: A Review

Anti-cancer Activity of Vitamin-C Against Leukemia: A Review

Call for Papers

Volume-8 | Issue-6

Last date : 27-Dec-2024

Best International Journal
Open Access | Peer Reviewed | Best International Journal | Indexing & IF | 24*7 Support | Dedicated Qualified Team | Rapid Publication Process | International Editor, Reviewer Board | Attractive User Interface with Easy Navigation

Journal Type : Open Access

First Update : Within 7 Days after submittion

Submit Paper Online

For Author

Research Area


Anti-cancer Activity of Vitamin-C Against Leukemia: A Review


Tarun Gupta | Virendra Kumar Vishwakarma | Manoj Kumar Maurya | Khushdil Khan

https://doi.org/10.31142/ijtsrd14250



Tarun Gupta | Virendra Kumar Vishwakarma | Manoj Kumar Maurya | Khushdil Khan "Anti-cancer Activity of Vitamin-C Against Leukemia: A Review" Published in International Journal of Trend in Scientific Research and Development (ijtsrd), ISSN: 2456-6470, Volume-2 | Issue-4, June 2018, pp.1264-1272, URL: https://www.ijtsrd.com/papers/ijtsrd14250.pdf

Leukemia is a gathering of malignancy began from bone marrow and will deliver impact on white platelet which will thus lead to anomaly. Presently the development of impact will continue by the undeveloped and juvenile WBC. Leukemia is an expansive term covering a range of infections. The focal point of enthusiasm for oncology especially in leukemia vitamin C has the best technique by which it demonstrates the more noteworthy outcome. An undeveloped cell takes amazingly elevated amounts of Ascorbic acid, which at that point allot their capacity and subdue the improvement of leukemia. Vitamin C levels manage HSC numbers and capacity. At the point when ascorbate levels are reduced, it can give the power to the deactivation of a catalyst called Tet2. The inactivation of TET2 is represented by change and insufficiency of vitamin-C may constrain Tet2 work in tissues in a way that builds the danger of leukemia. TET2 function is actually strengthen by ascorbate level. By encouraging DNA demethylation, tremendous-dose of ascorbate treatment can persuade stem cells to develop and the growth of cancerous stem cells will retarded by the tremendous dose of ascorbate level as investigated in mice. Tremendous dose of ascorbate might become a safe treatment for blood diseases caused by TET2-deficient leukemia stem cells, most likely in combination with other targeted therapies.

Leukemia, Blast cells, Vitamin-C, Tet2, DNA Methylation


IJTSRD14250
Volume-2 | Issue-4, June 2018
1264-1272
IJTSRD | www.ijtsrd.com | E-ISSN 2456-6470
Copyright © 2019 by author(s) and International Journal of Trend in Scientific Research and Development Journal. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (CC BY 4.0) (http://creativecommons.org/licenses/by/4.0)

International Journal of Trend in Scientific Research and Development - IJTSRD having online ISSN 2456-6470. IJTSRD is a leading Open Access, Peer-Reviewed International Journal which provides rapid publication of your research articles and aims to promote the theory and practice along with knowledge sharing between researchers, developers, engineers, students, and practitioners working in and around the world in many areas like Sciences, Technology, Innovation, Engineering, Agriculture, Management and many more and it is recommended by all Universities, review articles and short communications in all subjects. IJTSRD running an International Journal who are proving quality publication of peer reviewed and refereed international journals from diverse fields that emphasizes new research, development and their applications. IJTSRD provides an online access to exchange your research work, technical notes & surveying results among professionals throughout the world in e-journals. IJTSRD is a fastest growing and dynamic professional organization. The aim of this organization is to provide access not only to world class research resources, but through its professionals aim to bring in a significant transformation in the real of open access journals and online publishing.

Thomson Reuters
Google Scholer
Academia.edu

ResearchBib
Scribd.com
archive

PdfSR
issuu
Slideshare

WorldJournalAlerts
Twitter
Linkedin