Page 95 - Combine
P. 95

International Journal of Trend in Scientific Research and Development (IJTSRD) @ www.ijtsrd.com eISSN: 2456-6470
         [9]   Guest  T.  M.,  Ramanathan  A.  V.,  Tuteur  P.  G.  et  al.   [17]   Matsuda  N.,  Hattori  Yu.  Systemic  inflammatory
              Myocardial injury in critically ill patients: a frequently   response   syndrome   (SIRS):   Molecular
              unrecognized  complication.  J.A.M.A.  1995;  273:    pathophysiology and gene therapy. J. Pharmacol. Sci.
              1945—1949.                                            2006; 101: 189-198.
       [10]   Turner A., Tsamitros M., Bellomo R.Myocardial cell   [18]   Kaye A.D., Hoover J. M., Belukh A. R. Modern review of
              injury  in  septic  shock.  Crit.  Care  Med.  1999;  27:   multiple organ failure. Middle East J. 2005; 18: 273-
              1775—1780.                                            292.
       [11]   Ammann  P.,  Fehr  T.,  Minder  E.  I.  et  al.Elevation  of   [19]   Rudiger A., Singer M. Mechanisms of sepsis-induced
              troponin  I  in  sepsis  and  septic  shock.  Intens.  Care   cardiac dysfunction of thion. Crete. Care Med. 2007;
              Med. 2001; 27: 965—969.                               35: 1599-1608.
       [12]   Le Gall J.R., Lemeshow S., Saulnier F. A new simplified   [20]   Piper H. M., Schwartz P., Spar R., et al.Early release of
              acute  physiology  score  (SAPS  II)  based  on  a    enzymes from myocardial cells is not associated with
              European/North American multicenter study. J. A. M.   irreversible cell damage. Cell. Cardiol. 1984; 16: 385-
              A. 1993; 270: 2957-2963.                              388.
       [13]   Wu A. H. Increased troponin in patients with sepsis   [21]   Wu A. H., Ford L. Release of cardiac troponin in acute
              and septic shock: myocardial necrosis or reversible   coronary  syndromes:  ischemia  or  necrosis?  Klin.
              myocardial depression? Intens. CareMed. 2001; 27:     Chim. Acta 1999; 284: 161-174.
              959-961.
                                                              [22]   Babuin L., Jaffe A. S. Troponin: a biomarker of choice
       [14]   Wong  P.,  Mussay  S.,  Ramsewak  A.  et  al.Increased   for detecting heart damage C. M. A. J. 2005; 173: 1191-
              cardiac troponin T levels in patients without acute   1202.
              coronary syndrome. Postgrad. Med. 2007; 83: 200-  [23]
              205.                                                  Wu A. H. Increased troponin levels in patients with
                                                                    sepsis  and  septic  shock:  myocardial  necrosis  or
       [15]   Ammann P., Maggiorini M. Troponin as a risk factor    reversible  myocardial  depression?  Intensive  Care
              for  mortality in critically ill patients  without acute   Med. 2001; 27: 959-961.
              coronary  syndromes.  Am.  Coll.  Cardiol.  2003;  41:   [24]
              2004—2009.                                            Ver Elst K. M., Spapen H. D., Nguyen D. N. et al. Cardiac
                                                                    troponins  I  and  Ii  T-biological  markers  of  left
       [16]   Afessa  B.,  Green  B.,  Delke  I.,  Koch  K.  Systemic   ventricular dysfunction in septic shock. The wedge.
              inflammatory response syndrome, organ failure and     Chem., 2000; 46: 650-657.
              outcome in critically ill obstetric patients treated in
              the intensive care unit. Chest 2001; 120: 1271-1277.
















































        ID: IJTSRD40034 | Special Issue on Innovative Development of Modern Research                      Page 90
   90   91   92   93   94   95   96   97   98   99   100